How to Reverse Diabetes, and How Insulin May Accelerate Death. By Dr. Mercola. Great Britain, like the United States, has seen a remarkably rapid rise in pre- diabetes and type 2 diabetes over the last decade. According to a recent BBC News. British adults are now pre- diabetic. By 2. 01. 1, that figure had more than tripled, reaching 3. ![]() ![]() ![]() Researchers warn that this will lead to a massive avalanche of type 2 diabetics in upcoming years, which will have serious consequences for health care and life expectancy. What's worse, both type 1 and type 2 diabetes among children and teens has also skyrocketed. Incidence of type 2 diabetes among children aged 1. Conventional Medicine Has It All Wrong.. First, it tells us that diabetes cannot be primarily caused by genetics, and secondly, it literally screams that something we're doing, consistently and en masse, is horribly wrong, and we need to address it. Unfortunately, Dr. Diabetes: WHO health topic page on diabetes provides links to descriptions of activities, reports, publications, statistics, news, multimedia and events, as well as. Great Britain, like the United States, has seen a remarkably rapid rise in pre-diabetes and type 2 diabetes over the last decade. Related Articles: Is there a Indian vegetarian version of the GM Diet The GM General Motors Diet India Vegetarian version. GM Diet Indian Version The GM diet is. Although she was but forty-five, some obscure disease had taken the fire out of her figure. MSN Health and Fitness has fitness, nutrition and medical information for men and women that will help you get active, eat right and improve your overall wellbeing. Ron Rosedale wrote in 2. D. I. E from their flawed prescriptions, which stem from a basic lack of insight into the root cause of this disease. D. I. E., here, is a clever acronym for . The reality is that diabetes is a disease rooted in insulin resistance. This is why the medical community's approach to its treatment is not getting anywhere. Treating type 2 diabetes with insulin is actually one of the worst things you can do.. Recent research has come to the same conclusions that Dr. Rosedale warned us about nearly a decade ago, which is that treating type 2 diabetes with insulin can lead further to the development of type 1 diabetes. And, not only are conventionally- trained doctors wrong about the cause of the disease, but they continue to pass along seriously flawed nutritional information as well, which allows the disease to increase to epidemic proportions. Definitions of Terms. Before we get into the nitty- gritty of causes and treatments for diabetes, let's clarify the difference between type 1 and type 2, and the emergence of what some are now referring to as . It is conventionally diagnosed by having a fasting blood sugar between 1. Pre- diabetes is very easy to turn around. Simply swapping processed foods for whole organic foods lower in sugar and sugar- forming carbohydrates combined with a few minutes of daily exercise will quickly put you on the road to reversing this condition. As your insulin resistance progresses, your liver makes too much sugar and fat, and your skeletal muscles are less able to burn them and make glycogen, which is how glucose is stored in your muscles and liver. In turn, there is an increase in sugar and fats in your bloodstream which leads to high triglyceride levels and increased body fat- -especially abdominal fat, and higher blood pressure. Traditionally, type 1 diabetes develops before the age of 2. It used to be relatively uncommon, but as noted above, its incidence is rapidly rising. There is currently no known way to completely reverse this. For example, Columbia University scientists claim that by turning off a particular gene, human gut cells can be converted into cells that produce insulin in response to dietary sugar. In type 2 diabetes, the pancreas is producing some insulin, in fact usually too much, but is unable to recognize the insulin and use it properly. This is an advanced stage of insulin resistance, which is typically caused by a diet that is too high in sugars and sugar- forming foods. While anyone can get type 2 diabetes, you are typically considered at highest risk if you are overweight, sedentary, if you are a woman who had gestational diabetes, have family members with type 2 diabetes, or have metabolic syndrome. However, all of these really have the same underlying root of insulin and leptin resistance. Rosedale has been saying for two decades, that insulin therapy in type 2 diabetic patients may indeed do more harm than good. As reported by Medical News Today: 9. The higher A1c levels are, the greater the risk of other health problems. Sometimes the condition can be managed through changes in diet, but other patients with type 2 diabetes may need medication - such as insulin or metformin - to help lower their blood sugar levels, and ultimately, reduce the risk of diabetes complications. But the researchers of this latest study.. But for a patient who starts treatment for type 2 diabetes at age 7. ![]() The researchers say this prompts the question - is 1. Alzheimer's disease was tentatively dubbed . This extremely helpful guide, called the “Fatty Liver Diet Guide” is an ebook that deals with every aspect and ramification of being diagnosed with fatty liver. What to do After a Cancer Diagnosis. Griseofulvin Cured Man's Colon Cancer. Griseofulvin is an anti fungal medication and much science exists that many. Sounds frustrating, Richard W. Since your diet is low in fat with zero animal fats, diet most likely isn’t the problem. However, you may still have some fat in your. Your brain also produces insulin, and this brain insulin is necessary for the survival of your brain cells. A drop in insulin production in your brain may contribute to the degeneration of your brain cells, and studies have found that people with lower levels of insulin and insulin receptors in their brain often have Alzheimer's disease. ![]() Researchers have now discovered that insulin does far more than simply regulating blood sugar. Your brain does not require glucose, and actually functions better burning alternative fuels, especially ketones. In fact, Dr. Rosedale believes that it is the constant burning by the brain of glucose that is primarily to blame for Alzheimer's and other brain disorders Insulin is actually a . This is why reducing the level of insulin in your brain impairs your cognition. Other research. 11 shows that type 2 diabetics lose more brain volume with age than expected—particularly gray matter. This kind of brain atrophy is yet another contributing factor for dementia. As reported by Medical News Today: 1. There is no cure.. As you over- indulge on sugar and grains, your brain becomes overwhelmed by the consistently high levels of glucose and insulin that blunts its insulin signaling, leading to impairments in your thinking and memory abilities, eventually causing permanent brain damage. Additionally, when your liver is busy processing fructose (which your liver turns into fat), it severely hampers its ability to make cholesterol, an essential building block of your brain that is crucial for optimal brain function. Indeed, mounting evidence supports the notion that significantly reducing fructose consumption is a very important step you can take to prevent Alzheimer's disease. Root Causes of Type 1 Diabetes. Contrary to type 2 diabetes, type 1 is not may not be rooted in insulin and leptin dysfunction caused by excessive sugar (and carbohydrate) consumption. However, over the past several years, research has given us important clues about its predisposing conditions. Two important ones that you have more or less complete control over are. Research suggests that sun avoidance may play a major role in the development of insulin dependent diabetes. The further you move away from the equator the greater your risk of being born with, or developing type 1 diabetes. A major key to preventing type 1 diabetes in children is to ensure that pregnant mothers have optimal vitamin D stores. There is also strong evidence that this can decrease your child's risk of autism. Once your child is born, ensuring he or she gets optimal sun exposure (and/or wise use of oral vitamin D supplementation) could virtually eliminate the risk for type 1 diabetes. An excessive focus on a germ- free environment is another potential contributing factor that impairs immune function. In 2. 00. 8, animal research. There is a good deal of evidence that a contributor to the rising rates of type 1 diabetes is raising our children in too sterile an environment. Many parents religiously use antibacterial soaps and keep their children away from the natural dirt, germs, viruses and other grime of childhood. Antibiotics, which kill all of the good and bad bacteria in the gut, are also overused in childhood. The lesson here is, it's okay to let your child get dirty. Use plain soap and water for washing. Avoid antibiotics unless absolutely necessary, and feed them naturally fermented foods such as yogurt, pickles and sauerkraut. Root Causes of Insulin Resistance, Pre- Diabetes, Metabolic Syndrome, and Type 2 Diabetes. Type 2 diabetes involves loss of insulin and leptin sensitivity. This makes it easily preventable and nearly 1. One of the driving forces behind type 2 diabetes is excessive dietary fructose, which has adverse effects on all of metabolic hormones—including two key players: insulin and leptin. It's important to realize that even though fructose is relatively . So, while you may not notice a steep increase in blood sugar immediately following fructose consumption, it is likely changing your entire endocrine system's ability to function properly behind the scenes.. All carbohydrates that are not fiber will be quickly metabolized into sugar, and it makes little sense to eat large amounts of sugar to keep your blood sugar lower. Conventional medicine describes diabetes as a disease characterized by elevated blood sugar. The reality however is that type 2 diabetes is NOT the result of insufficient insulin production. It's actually the result of too much insulin being produced on a chronic basis primarily from eating the high carbohydrate, low fat diet recommended by the ADA and AHA to prevent and treat this. This also occurs with leptin. It's really important to realize that T2 diabetes is not caused by elevated blood sugar or . The root cause is insulin and leptin resistance which is why prescribing insulin is one of the WORST things you can do for type 2 diabetes, as it will actually worsen your insulin and leptin resistance over time. You do not need more insulin. You need to restore the sensitivity of your insulin and leptin receptors by keeping their levels low! A small amount is stored as a starch called glycogen, but the majority is stored as fat. Therefore, insulin's primary role is not to lower your blood sugar, but rather to store this extra energy as fat for future needs when food may not be available. The fact that insulin lowers your blood sugar is merely a . Taking more insulin just makes you fatter! The Normal A1. C Level - Diabetes Developments. You want to control your diabetes as much as possible. You wouldn’t be reading this if you didn’t. So you regularly check your A1. C level. This is the best measurement of our blood glucose control that we have now. It tells us what percentage of our hemoglobin — the protein in our red blood cells that carry oxygen — has glucose sticking to it. The less glucose that remains in our bloodstream rather than going to work in the cells that need it the better we feel now and the better our health will continue to be. As we are able to control our diabetes better and better, the reasonable goal is to bring our A1. C levels down to normal — the A1. C level that people who don’t have diabetes have. But before we can even set that goal, we have to know what the target is. The trouble with setting that target is that different experts tell us that quite different A1. C levels are “normal.” They tell us that different levels are normal — but I have never heard of actual studies of normal A1. C levels among people without diabetes — until now. The major laboratories that test our levels often say that the normal range is 4. They base that range on an old standard chemistry text, Tietz Fundamentals of Clinical Chemistry. The Diabetes Control and Complications Trial or DCCT, one of the two largest and most important studies of people with diabetes, said that 6. But the other key study, the United Kingdom Prospective Diabetes Study or UKPDS, which compared conventional and intensive therapy in more than 5,0. Those levels, while unsubstantiated, are close. But they comes along one of my heroes, Dr. Bernstein, the author of the key text of very low- carb eating for people with diabetes, Dr. Bernstein’s Diabetes Solution. Bernstein himself developed type 1 diabetes in 1. For my patients. Bernstein says is normal is so at odds with the other experts that at least a year ago I determined to find scientific proof of what a normal A1. C level actually is. It turned out to be a lot more difficult to find than I ever imagined. My personal quest for a normal A1. C level and that of my favorite Certified Diabetes Educator drove that search. When I learned in 1. I had diabetes and that my A1. C level was 1. 4. I was gradually able to bring it way down. Lately I have been doing everything I can think of to try to get my A1. C down to normal. But in 2. 00. 8 my level in nine separate A1. C tests always ranged from 5. That’s far from normal, according to Dr. Bernstein. My favorite Certified Diabetes Educator is also doing everything she can to get a normal A1. C level. And she doesn’t even have diabetes — which she double- checked by taking a glucose tolerance test — but her most recent A1. C was 5. 4. What could we be doing that is so wrong? Each of us is thin, eat a very healthy diet, exercise a lot, take care of our teeth and gums, which is a major source of infection. Could we have other infections or stresses that prevent us from getting our A1. C levels down to “normal”? It turns out that my favorite Certified Diabetes Educator and I have normal A1. C levels after all. I learned this just yesterday when I finally tracked down actual research determining what normal levels are. A friend suggested that I contact the people who run the standardization program for A1. C testing. This organization affiliated with the University of Missouri is the NGSP. Those initial used to stand for the National Glycohemoglobin Standardization Program. But now that the NGSP is international, they changed the name. So I called Curt Rohlfing, the NGSP data manager and technical writer/research analyst at the University of Missouri. And finally hit pay dirt in my quest for learning what a normal A1. C is. Curt told me that every three or four years his lab at the university studies a group of people who don’t have diabetes to scientifically determine what a normal A1. C level is. The results from one study to the next are always close, Curt told me. In their most recent study they tested 2. Missouri. I asked how they knew if the people they tested didn’t have diabetes. They ranged from 4. Curt replied. That’s at plus or minus 3 standard deviations. I am certainly no statistician. But Curt tells me that it includes about 9. The range is narrower — 4. This includes about 9. The upper limit is the more important one,” Curt explained further. In fact, levels below 4. Remember these are the ranges obtained by the people who set the standards for A1. C tests. Sadly, however, not every laboratory or home test kit meets those standards. Maybe the lab that Dr. Bernstein uses doesn’t. Curt suggests that you ask your doctor if the lab running the test uses a method that is certified by the NGSP. The first conclusion of the research for me is that we need to shoot for a normal A1. C level of no more than 6. However, an A1. C level of 6. That’s why the American Diabetes Associations sets the goal conservatively at 7. Still, a lower A1. C level among people who take those medications is possible without hypos. Bernstein has amply shown that both in his own life and that of thousands of his patients. And certainly, for those of us who don’t take insulin injections or one of the sulfonylureas we can set our goal even lower. That’s because we have to understand the different between normal and optimal. For example, two- thirds of all American adults are overweight. Thus it has become normal in our culture to be overweight. Likewise, the average American gets little exercise, and that is also normal. We know that being a chubby couch potato isn’t optimal.“I’m going to aim to be in the lower end of the normal A1. C range,” my favorite CDE tells me, “because that is what I believe is optimal for human health.” And now that I know my A1. C is in the normal range I am still going to do my best to bring it down as much as possible. Are you? This article is based on an earlier version of my article published by Health. Central. Never Miss An Update. Subscribe to my free newsletter “Diabetes Update”I send out my newsletter on first of every month. It covers new articles and columns that I have written and important developments in diabetes generally that you may have missed.
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